ACLS
To estimate atrial rate, count number of P waves over 6s period & multiply by ________.
10
ECG should be obtained & interpreted w/in how many mins of pt arrival if suspected of ACS
10 minutes
IV nitro for pulm edema or HTN should be titrated to maintain SBP ___% < baseline if normal BP (30 below in pts w. HTN)
10%
CPR Success Rate
10% for out of hospital - 20% for in hospital
Member of resuscitation team is preparing to admin meds IV to pt in cardiac arrest. Team member follows each med admin w. bolus of fluid. How much would team member give
10-20 mL fluid bolus.
When providing high-quality CPR on adult, what is proper rate for chest compressions?
100-120 bpm
How much time from pt arrival at facility should brain imaging be ordered for pt w. suspected stroke
10m
In ventricular tachycardia, QRS complexes are wide, lasting longer than:
120 milliseconds
Nurse is preparing to defibrillate pt in cardiac arrest w. biphasic defib & needs to set energy dose according to manufacturer's recommendations, which is usually
120-200 joules
Recommended energy dose for biphasic synchronized cardioversion of AFib
120-200J
Percentage of embolic strokes associated w. AFib
15-20%
Pt w. suspected stroke arrives at 1910 & stroke team ensures comprehensive neuro assessment using NIHSS is completed & brain imaging is performed by
1930
Chemical Colorimetric Analysis
pH-sensitive paper w/in chamber placed btw tracheal tube & ventilation device - Purple, tan, or yellow
Pt w. dizziness & SOB has HR 40, initial atropine was ineffective, & monitor/defibrillator not equipped w. transcutaneous pacemaker. What is appropriate dopamine dose to give
2-10 mcg/kg/min
When providing high-quality CPR on adult, what is proper depth for chest compressions?
2-2.4"
STEMI presents at 2130 & states s/s started ~2000. After confirming dx & initiating care, PCI scheduled. Facility capable of PCI. For best outcome, PCI performed by
2300 - Reperfusion w/in 1.5h of 1st medical contact if pt doesn't need to be transferred
Pt w ischemic stroke receives fibrinolytic therapy. Anticoagulant therapy should be withheld for how long
24 hours
Gold Standard for Successful Intubation
3 ETCO2 waveforms of equal height
Troponin levels should be measured at initial presentation & then __ hours later
3-6
Fibrinolytic therapy should initiated w/in _ min of 1st medical contact if primary PCI can't be performed w/in 90m of arrival
30
Initial dose of amiodarone for pt in cardiac arrest
300 mg IVP - Subsequent doses are 150 mg IVP
When performing high-quality CPR on adult w/o advanced airway in place, what is correct ratio of compressions to ventilations?
30:2
EKG reveals following rhythm, which feature would HCP interpret as indicating AFib
Absence of discrete P waves & presence of irregularly irregular QRS complexes
Team leader instructs team member to perform intervention. To practice good communication, team member should do which of the following? (3)
Acknowledge completion of intervention, use nonverbal communication methods to indicate follow through w. intervention, & acknowledge initiation of intervention
40yo in waiting room of PCP approaches staff & says, "I'm having really severe, crushing CP that's moving to both arms." Pt diaphoretic & dyspneic. Action appropriate for staff member to take
Activate emergency medical services system
Capnogram
Actual waveform
Contraindications to therapy w. Beta Blockers in pts w. ACS
Acute HF - Recent MI - Low CO - R/O cardiogenic shock
Which non-respiratory differential dx would you consider when assessing pt w. respiratory compromise?
Acute valvular insufficiency, cardiac tamponade, cardiogenic pulmonary edema
Cardiac monitoring of pt in cardiac arrest reveals VFib. Team should immediately
Administer 1 shock
In addition to resuming CPR, what other actions should the team take at this time
Administer epinephrine. Consider establishing an advanced airway. Consider underlying causes (Hs and Ts).
Nitro Dosage
Administer nitro 0.4 mg q5m by SL tablet or spray (1-3x) to relieve ischemic CP
Initial interventions appropriate for primary assessment of pt who has achieved ROSC
Assess neuro status - Establish noninvasive BP monitoring - Assess LOC - Consider placement of ET tube to support mechanical ventilation
Actions from prehospital providers that can improve outcomes for acute stroke pts
Assigning highest priority to potential stroke calls - Notifying receiving facility in advance of pt arrival - Transporting to facility capable of assessing & treating stroke
Team Leader Responsibilities
Assigns & understands team roles - Sets clear expectations - Prioritizes, directs & acts decisively - Encourages/allows team input & interaction - Focuses on big picture - Monitors performance while providing support - Acts as role model - Coaches team - Re-evaluates & summarizes progress - Leads debriefing
Where do We Measure ETCO2
End of expiration (end of phase 3) or D in picture
Normal Respiratory Baseline
Flat & consistent from breath to breath
On capnography waveform, what finding indicates pt isn't rebreathing CO2?
Flat Baseline
Normal Capnogram Consists of:
Flat baseline, steep upstroke/downstroke, flat plateau, & ETCO2 btw 35-45 mmHg
Tamponade (Cardiac)
Fluid around heart compresses heart (can't fill or pump) - Decompress via removal of fluid in pericardium
Myocardial Stunning Tx
Fluids to optimize volume status - Dobutamine 5-10 mcg/kg per min - Mechanical augmentation (IABP)
Primary assessment
Focused assessment of airway, breathing, circulation, disability, & exposure (ABCDE) to identify potentially life-threatening conditions & address them immediately
Pt w. acute renal failure has cardiac arrest. Just before arrest, ECG had peaked T-wave. Possible cause of arrest
Hyperkalemia - Suspected in all pts w. acute/chronic renal failure w. wide-complex ventricular rhythm or tall, peaked T waves on ECG before arrest
Pt comes w. suspected stroke. Pt AxO w. family who says, "I noticed he was slurring his words & had trouble walking, like his leg was numb." What finding suggests pt is experiencing condition that mimics stroke
Hypoglycemia
Prior to cardiac arrest, EKG had flat T waves, prominent U waves & prolonged QT interval. What cause suspected
Hypokalemia
Team is providing care to pt experiencing VTach & serum electrolyte levels are contributing c/o current condition. Which electrolyte imbalance(s) would most likely be involved
Hypomagnesemia - Hypocalcemia
Which findings may suggest pt is experiencing hemodynamic compromise c/b bradyarrhythmia? (5)
Hypotension - Ischemic CP - S/S of shock - AMS - Acute HF
Physical exam findings in pts w ACS may include which conditions
Hypotension - Pulmonary edema - Crackles
Metabolic Causes for Decreased CO2
Hypothermia - Increased MM Relaxation - Increased Depth of Anesthesia
Gradual CO2 Increase C/B
Hypoventilation - Laparoscopic insufflation - Increased Metabolism - Increased Pulmonary perfusion
When a patient is in cardiac arrest, it is important to consider reversible underlying causes. What underlying causes should the team consider
Hypovolemia - Hypoxia - Hypothermia - Acidosis - Potassium imbalance - Toxins - Cardiac tamponade - Tension pneumothorax - PE - MI
EKG shows tachyarrhythmia. Pt hemodynamically stable w. HR 120-135. Based on secondary assessment, what statement by pt would team interpret as possible contributing causes
I've had a terrible cold w. horrible cough/fever over past wk - I've been anxious lately bc I just lost my job - I've been vomiting for 2d d/t a GI bug
After cardiac arrest & ROSC, pt unable to follow verbal commands so TTM initiated. Which method(s) would be appropriate
Ice-cold IVF bolus - Applying cooling blankets to pt body - Endovascular catheter
Benefits of ETCO2
Identifies early hypoventilation (dental procedures, GI procedures in morbidly obese, & PACU) - Shows effectiveness of CPR/ventilatory rate - Monitor high dose opioid/hypnotic drug - Confirm ETT placement - Guide vent mgmt
Resuscitation team responds to in-hospital arrest, immediate actions
Identify rhythm - Ensure high-quality CPR being performed
Pt w. s/s of ACS & ST elevation in II, III, aVF. Next action
If ECG = inferior wall MI, 15-lead to screen for R ventricular infarction
Inspiratory downslope
Phase of capnography waveform representing inhalation. Inhalation occurs & CO2 is rapidly purged from airways & alveoli.
Respiratory Baseline
Phase on capnography waveform representing beginning of exhalation. During phase, "dead space" air (i.e. air in airways from bronchioles to nasal cavity that doesn't contain CO2) is exhaled from body.
Respiratory upstroke
Phase on capnography waveform representing exhalation of air containing CO2 from alveoli. For most pts, respiratory upstroke should be nearly vertical.
Which diagnostic tests used for monitoring perfusion status in post-cardiac arrest pt
Serum lactate, if cells are adequately perfused and/or oxygenated it won't be produced - Start metabolizing inefficiently = Producing more acid byproducts
Pulseless VTach is Treated the Same As
VFib
Shockable Rhythms
VFib - Pulseless VTach - Don't shock asystole/PEA - As soon as shockable rhythm seen, administer defib
Pt being treated determined to have NSTE-ACS, early invasive tx planned d/t what finding
VTach
EKG shows QRS complex w. regular rhythm = Narrow-complex SVT. Pt hemodynamically stable. Tx initiated 1st
Vagal maneuvers
STEMI pt experiencing CP refractory to SL Nitro prescribed IV Nitro. When administering med, titrate to maintain SBP of
90+
Ideal time frame for performing percutaneous coronary intervention (PCI) in pt w. STEMI
90m of pt arrival at facility
In pt showing s/s of respiratory compromise, provide minimum level of supplemental O2 needed to maintain O2 saturation of at least:
94%
Provide minimal level of supplemental oxygen needed to maintain oxygen saturation of at least _____
94%.
Pt w. suspected ACS c/o SOB & given supplemental O2 - HCP determines O2 dose correct based on what SaO2 level
95%
35yo female ECG is consistent w. STEMI = New ST elevation at J point in leads V2 and V3 of at least which size
0.15 mV
Nitro dose (sublingual tablet or spray) to be administered to pt suspected of having ACS
0.4 mg q5m
Ischemic stroke pt arrives at 0200 & s/s started at ~0030. After completing necessary assessments, team dx ischemic stroke. Pt determined candidate for fibrinolytic therapy. To achieve best outcomes, team should initiate therapy no later than
0300
Pt in cardiac arrest & metabolic acidosis determined to be underlying cause, initial dose of sodium bicarb
1 mEq/kg
When advanced airway in place & pt in cardiac arrest, compressions & ventilations delivered continuously w. no interruptions. 1 provider delivers __________ q6s while 2nd provider performs compressions at rate of 100-120/min
1 ventilation
Resuscitation team debriefing following pt cardiac arrest where ACLS was initiated. Pt required placement of advanced airway to maintain patency. Statement indicates team performed high-quality CPR
1 ventilation q6s & 100-120 compressions/min
After 3 shocks, admin lidocaine for cardiac arrest. What is dose?
1-1.5 mg/kg, then 0.5-0.75 mg/kg q5-10m, max dose 3 mg/kg
Post-cardiac arrest pt has SBP < 90 mmHg. What interventions should be considered next?
1-2L IV isotonic crystalloid fluid bolus - Vasopressor infusion w. epinephrine (0.1-0.5 mcg/kg/min)
You'd expect to see ETCO2 < ___ in presence of hyperventilation
35 mmHg
HCP initiates ventilations to ensure adequate breathing/oxygenation. During ventilations, capnography established to evaluate adequacy of ventilations. HCP determines ventilations adequate based on which ETCO2 value
35-45 mmHg
What end-tidal carbon dioxide (ETCO2) value confirms adequate ventilation in intubated pt w. respiratory arrest?
35-45 mmHg
Ideal time frame for fibrinolytic therapy admin
3h from s/s onset
Reversible causes of cardiac arrest
4 H's = Hypovolemia, Hypoxia, H+ (acidosis), Hypo/hyperkalemia, Hypothermia
How much time from pt arrival at facility does brain imaging have to be interpreted
45 min
20yo man w. respiratory depression brought in by parents. Parents state, "[They] found him w. various needles & syringes around him, but have no idea what he took." Opioid OD suspected & initial dose of naloxone administered at 2200. Pt doesn't respond to initial dose. Team would expect to administer 2nd dose after how many mins
4m
Pt experiencing respiratory distress secondary to exacerbation of COPD & begins exhibiting s/s of worsening respiratory function & experiences respiratory arrest. Team intervenes, delivering ventilations via BVM delivering 1 ventilation at which interval
5-6s
Which statement about electrocardiography is true?
5-lead ECG uses 4 limb leads & 1 precordial lead to provide 7 views of electrical activity of heart
What ETCO2 value would you expect to see in presence of hypoventilation?
50 mmHg
R/O Torsades de Pointes increased when corrected QT interval (QTc) > ___ milliseconds
500
Sinus bradycardia is identical to normal sinus rhythm, except rate is less than _____ bpm
60
Ideal time frame for administration of endovascular therapy
6h from s/s onset
Bag ventalition device holds ______mL
700
About __% of pts w. ACS each yr, the arterial occlusion results in UA or NSTEMI & remaining __% result in STEMI
70; 30
Abnormal finding in 1 Cincinnati Prehospital Stroke Scale assessment areas is associated w. what stroke probability
72%
Proper ventilation rate for pt in cardiac arrest w. advanced airway in place
8-10 breaths/min
Primary Assessment
ABCDE approach & provide care PRN - Ensure patent airway - Assist ventilation PRN - Pulse ox, capnography, & cardiac monitoring - Provide minimum supplemental O2 to maintain 94% saturation (88-92% in pts who rely on hypoxic drive) - Adjust ventilations PRN to maintain ETCO2 35-45 mmHg - Establish vascular access & prepare to provide CPR/Defibrillation if condition deteriorates
Hover hands over chest but don't touch during:
AED shock (to minimize time not performing compressions)
Stroke team assessing suspected stroke, pt alert & able to carry on convo, although pt has difficulty getting words out. Testing confirms pt had ischemic stroke. Hx of which arrhythmia would alert team to pts increased r/o stroke
AFib
Synchronized cardioversion indicated for tx of which arrhythmias? (3)
AFib - Monomorphic VTach w. regular rhythm - AFlutter
During primary assessment, what tool would you use to determine pts LOC?
AVPU Model
30yo brought in cardiac arrest. Cardiac monitor shows VTach. Possible precipitating factors of VFib
Electrocution, myocardial ischemia/infarction, shock, stimulant OD, VTach
Recognize
After you gather assessment data, use critical thinking, your past clinical experience and your general knowledge to correctly interpret the meaning of the data and gain an understanding of the patient's clinical situation and care needs. This understanding enables you to determine your next steps.
NGT After Arrest or Advanced Airway Placement Rationale
Air fills in stomach & presses on lungs - Pt often recently ate/drank & at r/o aspiration
In inflammatory conditions, waveform may still be square, despite narrowing of airway, bc
Alveoli are still empty at same rate
Which anatomic landmark should be used to ensure proper placement of precordial electrodes for 12-lead ECG?
Angle of Louis (sternal angle), which is adjacent to 2nd rib. Then palpate down along sternal border to identify 4th ICS
You need to obtain intraosseous (IO) access. Identify preferred site.
Anterior proximal tibia often used bc it provides flat surface w. relatively thin outer layer of bone, large marrow cavity, & easily identifiable landmarks to facilitate placement. Proximal humerus also used.
Key questions to answer in assessment/mgmt of post-cardiac arrest pt
Any potentially reversible underlying c/o cardiac arrest that need to be addressed - Is pt candidate for TTM - Does pt require reperfusion therapy to address MI
Oxygen-hemoglobin dissociation curve depicts relationship btw partial pressure of oxygen (PaO2) & the:
Arterial oxygen saturation
Changes in what can affect rate & depth of breathing? (3)
Arterial pH - Arterial O2 levels - Arterial CO2 levels
Pt c/o SOB has long hx of COPD. Assessment reveals respiratory failure. Initial priority
Assisted ventilation w. BVM - Pts who can't ventilate adequately despite open airway or have insufficient respiratory effort require assisted ventilation initially w. BVM
In _________, atrial contraction occurs at such a rapid rate that discrete P waves separated by flat baseline can't be seen.
Atrial Flutter
In event of primary pacemaker dysfunction/failure, what part of heart's conduction system can function as backup pacemaker?
Atrioventricular (AV) Node
What part of heart's electrical conduction system helps to protect ventricles from atrial tachyarrhythmias?
Atrioventricular (AV) node
What is 1st-line therapy for unstable pt w. symptomatic bradycardia?
Atropine
Pt w. SOB/AMS arrives, team completes assessments, & cares for pt including cardiac monitoring/pulse ox, supplemental O2, ensuring adequate ventilation, & obtaining IV. Team reviews EKG (figure). Med most likely to administer
Atropine 0.5 mg q3-5m
Pt presents w. new onset of dizziness & fatigue. HR 35, BP 70/50, RR 22, & 95%. What is 1st med given
Atropine 0.5mg
O2 Warning During Defibrillation
Be sure O2 isn't blowing over pt chest during shock
Mr. Hernandez is found to be unresponsive, with no pulse and no breathing. What is the appropriate next intervention
Begin CPR and administer one shock.
Pt w. ACS is experiencing cardiogenic shock. Which adjuvant therapy would be contraindicated
Beta Blockers
Modern Defibrillators
Biphasic & 200J
Pathophysiologic SE of cardiac arrest
Brain injury - Systemic dysfunction - Myocardial Stunning (dysfunction)
Secondary assessment
Broader assessment to narrow list of differential dx & discover underlying causes. Includes focused hx, exam, & diagnostic testing
CO2 Facts
Byproduct of cellular metabolism - Transported by Hgb to lungs - Eliminated from alveoli (during ventilation)
Which blood tests are often indicated in evaluation of pts experiencing cardiovascular, cerebrovascular or respiratory emergency?
CMP, Toxicology, CBC, Cardiac markers, ABGs, Blood glucose
Flat Baseline Indicates
CO2 completely purged from airway
Bradycardia Requires Tx if
CP or SOB present - Tx = 0.5 mg Atropine q3-5m, max 3mg - Other Tx = Transcutaneous pacing, Dopamine 2-10 mcg/kg/min - Epi 2-10 mcg/min
Dx test typically obtained at presentation to r/o other c/o acute CP & to identify pulmonary congestion
CXR
Indications of Rebreathing CO2
Can occur w. faulty expiratory valve or exhausted absorber system - Inspiratory CO2 consistently > 0 - Insufflation of CO2 into airway from ENT - Will likely not see increase in 3 waveforms
Primary assessment tasks if suspected acute stroke
Cardiac monitoring - NPO - Supplemental O2 if pulse ox < 94%
The team resumes CPR, delivers a third shock and administers amiodarone, 300 mg IV push. At the next rhythm check, the monitor displays this rhythm. What action should the team take next
Check for a pulse.
Based on visual survey, pt appears to be unresponsive. What should your immediate next action be?
Check for responsiveness using shout-tap-shout sequence If they're unresponsive, call for additional resources & simultaneously check breathing/pulse for 5-10 seconds
What is the appropriate next action for the team to take at this time
Check for responsiveness, breathing and a pulse.
Which assessments would be included in primary assessment?
Checking for life-threatening injuries & checking airway patency
Team member confirms message is received & understood. What communication technique is team member using?
Closed-Loop Communication
Closed-loop communication
Communication technique used to prevent misunderstandings; receiver confirms message has been received & understood.
Flat Expiratory Plateau Indicates
Complete exhalation/emptying of CO2 from alveoli - ETCO2 reading accurate & at peak
If capnography is < 10 there may be probelm with _________
Compressions
Crew Resource Management
Concept helps to promote effective/efficient teamwork & reduce likelihood of errors. Emphasizes using all available resources (people, equipment, procedures) to reduce likelihood of human error & promote effective/efficient teamwork. Guides team members to communicate directly & effectively w. team leader about dangerous or time-critical decisions. When problem arises, team members must get attention of team leader, state concern, describe problem as they see it & suggest solution. Team leader provides direction, enabling team to work together to resolve issue.
You have just placed advanced airway. How should you verify correct placement?
Confirm initially, if pt moved & ongoing basis. Methods: Physical assessment (observing for bilateral chest rise & auscultating over lungs/epigastrium) & confirmation tool (i.e. capnography) Fogging in tube & improved O2 saturation on pulse oximetry aren't reliable methods of confirmation
Cardiac monitor indicates VTach. Pt has pulse & not showing s/s of hemodynamic compromise. ECG reveals irregular rhythm w. QRS complexes > 0.12s in duration. Which action would be appropriate
Consider antiarrhythmic infusion & expert consult
Most reliable method of confirming & monitoring correct placement of ET tube
Continuous waveform capnography
Pt has CP after Nitro x 3, cardiac biomarkers elevated, & ST-segment depression. Consider
Coronary angiography - For high-risk NSTE-ACS, early invasive strategy w/wo revascularization should be considered - Refractory ischemic CP is indication
When auscultating lung sounds in pt w. left ventricular dysfunction, you'll hear
Crackles
Important questions regarding medical hx to ask pt w. suspected stroke
Current meds & when s/s began
Which underlying mechanism can lead to hypoxia in pt w. right shift of oxygen-hemoglobin dissociation curve?
Decreased affinity for oxygen binding
Don't Intubate, Start IV, Etc. Until You
Defibrillate
Assess, Recognize & Care
Describes ongoing process of gathering data about pt condition, using data to identify problem & intervening to address problem. Acutely ill pt condition can change rapidly (for better or worse), you must continuously assess pt, recognize whats happening & provide care accordingly
Urine Output & Serum Creatinine Rationale
Detect AKI - Maintain euvolemia - Renal replacement PRN - Low output = Shock
Goals of secondary assessment in pt w. suspected arrhythmia? (2)
Determining potentially reversible causes of arrhythmia & determining the severity of s/s
Respiratory: What is body's primary muscle of inspiration?
Diaphragm
Which would be important to note when reviewing hx of pt in cardiac arrest
Disorders/Situations that could predispose pt to developing Hs/Ts - Medication use - Changes in pt clinical condition prior to arrest - R/F for cardiac or pulmonary conditions
Stroke pt is admitted to dedicated stroke unit, reflecting which 8 Ds of stroke care
Disposition
Expiratory Plateau
During this phase, the last of CO2-laden air from most distal alveoli exhaled from body. ETCO2 measured at end of exhalation (point D), representing peak level.
On capnography, absent expiratory plateau indicates:
Dynamic Hyperinflation aka auto-PEEP (auto-positive end expiratory pressure). Plateau loss produced by uneven alveolar emptying secondary to severe bronchospasm leading to air trapping.
Diagnostic study that should be delayed until ROSC
EKG
CO2 Goals
ETCO2 35-40 - ATCO2 40-45
Tan Colorimetric Analysis
ETCO2 4-15 mmHg (0.5-2% CO2)
Purple Colorimetric Analysis
ETCO2 < 4 mmHg (0.5% CO2)
Yellow Colorimetric Analysis
ETCO2 > 15 mmHg (> 2% CO2)
S/S of ROSC
ETCO2 goes up (abrupt increase, usually > 40) - HR/BP Increase - Spontaneous ventilation
Based on the primary assessment findings, what additional actions should the team take
Ensure vascular access and administer atropine. Administer supplemental oxygen.
Pt w. unstable bradyarrhythmia doesn't respond to atropine. Possible next tx
Epi/Dopamine infusion - Transcutaneous pacing
What drugs may be used as 2nd-line therapy for unstable pt w. symptomatic bradycardia?
Epinephrine - Dopamine
Absent CO2 C/B
Esophageal intubation (will see some ETCO2 initially then diminishes, why 3 equal height waveforms) - Death - Blocked sample line - Severe bronchospasm - Equipment malfunction - Hard cricoid pressure occluding tip of ETT
12-lead ECG shows wide-complex ventricular tachycardia in stable pt w. pulse. Which may be indicated in mgmt of pt?
Expert consultation and/or antiarrhythmic infusion
When providing transcutaneous pacing, appearance of wide QRS complexes & tall, broad T waves after each pacing spike confirms mechanical capture has been achieved. T/F?
False
Secondary Assessment
Goals are to discern underlying c/o respiratory compromise & evaluate severity of pt condition - Acute onset of respiratory distress is frequently pulmonary or cardiac in origin
Partial Disconnect
Gradual ETCO2 drop - Can also be from a cuff leak
Capnography
Graphic record of CO2 concentration
Team Member Responsibilities
Have necessary knowledge/skills to perform assigned role - Stay in assigned role but assist others PRN, as long as they're able to maintain own responsibilities - Communicate effectively w. team leader if they: Feel they're lacking any knowledge/skills to perform assigned roles, identify something team leader may have overlooked, or recognize dangerous situation/need for urgent action - Share info w. other team members - Focus on achieving goals - Ask pertinent questions & share pertinent observations - Participate in debriefing sessions - Crew
Capnogram Features
Height = CO2 - Frequency = RR - Normal baseline = 0 - Rhythm - Baseline - Shape
High-Risk vs Low-Intermediate Risk NSTE-ACS
High risk = Changes = Ischemia, i.e. ST depression/T-wave inversion, 2+ leads - Low/Intermediate-risk = No changes or nondiagnostic ST-segment or T-wave changes
Pt w. s/s of ACS has T-wave inversion in 2+ leads. To which clinical category should pt be assigned
High-risk NSTE-ACS
Rhythm check reveals shockable rhythm. When should chest compressions resume?
Immediately after rhythm check
Stroke S/S
Impaired balance - Sudden/Severe HA - Blurred vision
Circulatory/Respiratory Causes for Decreased CO2
Impaired peripheral circulation (insufflation, packing, positioning) - Decreased blood flow to lungs - Increased ead space - Hyperventilation (we do this to decrease ICP during neuro case & lap cases to blow off CO2)
Care
Implement appropriate care based on understanding of condition. Proper care can't be provided w/o effective assessment & accurate recognition of condition
6-Person High-Performance Resuscitation Team
Includes 3 who fulfill CPR/defib roles & 3 who perform leadership/supportive roles
How can we use PEEP to improve VQ mismatch
Increase PEEP little by little to decrease gradient as close to 1:1 as possible - Need A-line to get multiple ABGs
Equipment Causes for Increased CO2
Increased Apparatus dead space (smaller pt = bigger impact i.e. neonates) - Rebreathing - Obstruction in expiratory system (failed inspiratory/expiratory flutter valves cause us to rebreathe CO2. If inspiratory stuck open & expiratory side obstructed, exhalation can go up inspiratory side & be inhaled in next breath further increasing CO2 - If expiratory valve stuck open it allows exhalation to come back into pt when they start spontaneously breathing again
Circulatory/Respiratory Causes for Increased CO2
Increased CO - Hypoventilation - Upper airway obstruction - Rebreathing
Hyperventilation Can Cause
Increased dead space - Decreased height on capnography
Inotrope
Increases force of contraction
Therapeutic Hypothermia
Indwelling temp monitoring device - Prevent hyperpyrexia > 37.7 C - Induce if no contraindications - Cold IVF bolus 30 mL/kg - Surface/Endovascular cooling for 32-34 C x 24h - After 24h, slow rewarm 0.25 C/hr
Pt brought w. suspected opioid OD & in cardiac arrest. Priority action
Initiating high-quality CPR. arrest, Narcan is also recommended during arrest when opioid OD suspected but CPR takes priority over Narcan (Naloxone)
CP Descriptions associated w. ACS
Intermittent - Pressure/Squeezing/Tight/Aching/Heaviness - Radiates to arm(s) - Lasts > 3-5m
2 main types of strokes
Ischemic & Hemorrhagic
NIHSS Evaluates
Language deficits - Sensation & neglect - Cerebellar function - Motor & Visual Function - Level of consciousness
Equipment Causes for Decreased CO2
Leak in sample - Low sampling rate - High sampling rate - Cuff leak
Hyperventilation leads to alkalosis & a _______ shift of oxygen-hemoglobin dissociation curve.
Left
When Hgb's affinity for O2 is increased, O2 binds to Hgb easily, but offloading is difficult. This is reflected on oxygen-hemoglobin dissociation curve as shift to the _________.
Left
Pt brought in by spouse who says, "I think it's a stroke." Stroke team assesses pt using NIHSS. Area(s) included in assessment
Level of consciousness - Language deficits - Visual function
Relationship btw ETCO2 & PaCO2
Linear, ETCO2 measurement doesn't replace PaCO2 bc gradient can vary in size & direction
If Pt Isn't Perfusing
Little CO2 will be expelled & when pt starts perfusing more CO2 will be expelled (s/s of ROSC)
Normal Capnogram
Look like top hat - Y-Axis= mmHg - X-Axis= time - Phase 1= Inspiration - Phase 2= Beginning of expiration - Phase 3= Expiration - Phase 4= Beginning of inspiration
Pt has s/s suggestive of ACS has T-wave inversion < 2 mm, what category would you assign pt
Low/Intermediate-risk NSTE-ACS
Metabolic Causes for Increased CO2
MH (ETCO2 rises before temp) - Hyperthermia - Shivering/Convulsion - Pain - Catecholamines - Blood & Bicarb - Release of clamp/tourniquet - Absorption from laparoscopy - TPN
Acceptable brain imaging options for suspected acute stroke
MRI - CT w. or w/o - Diffusion-weighted MRI
What medication should be considered for Torsades de Pointes
Magnesium Sulfate
After fibrinolytic therapy pt transferred to monitored bed on stroke unit. Care focused on
Managing HTN - Controlling BG - Assessing for s/s of intracranial bleeding - Supporting airway, breathing, circulation
Capnometry
Measurement & numeric display of partial pressure or gas concentration of CO2
Increases/Decreases in ETCO2 are reliable in what pts?
Mechanically ventilated patients - Spontaneous breathing allows for changes in RR that compensate for changes in ETCO2 - Mechanically ventilated pts can't spontaneously adjust their RR
Healthcare provider conducting secondary assessment of pt w. arrhythmia. Which questions should provider seek answers to while obtaining pt hx? (3)
Meds currently being taken, what pt was doing when s/s began, & hx of pulmonary/thyroid disease
Brown lead going right side of chest in __________
Middle (~3-4th ICS)
CPR: Which statements apply to providing high-quality chest compressions? (3)
Minimize interruptions, proper depth, proper rate
Therapeutic Hypothermia Rationale
Minimizes brain injury & improves outcome (decrease brain's O2 consumption, decrease brain cell swelling)
As team leader, Dr. Hudson is responsible for monitoring CPR quality. Which of the following actions are necessary to ensure high-quality CPR
Minimizing interruptions to compressions. Providing compressions that are at least 2 inches (5 centimeters), but not more than 2.4 inches (6 centimeters), deep. Providing compressions at a rate of 100 to 120 per minute. Avoiding excessive ventilations.
What is capnography waveform graphical representation of?
Movement of CO2 through respiratory system
New ST-segment elevation in 2+ contiguous leads on 12-lead ECG suggests
Myocardial infarction
ECHO Helps Dx
Myocardial stunning - Wall-motion abnormalities - Structural problems - Cardiomyopathy - Decreased EF
Which airway device is most appropriate as initial intervention for semiconscious pt who requires ventilation w. BVM resuscitator?
NPA (nasopharyngeal airway) may be used w. conscious, semiconscious or unconscious pts
Conditions included in category of NSTE-ACS
NSTEMI & Unstable angina
Main difference btw NSTEMI & UA in NSTE-ACS
NSTEMI have elevated serum cardiac markers & pts w. UA don't
ECG finding diagnostic for STEMI in man 40+ yo
New ST elevation at J point in leads V2 & V3 that's at least 0.2 mV (≥ 2 mm)
Meds that should be administered to pt w. s/s of MI or infarction
Nitroglycerin & aspirin
Low/Intermediate-risk NSTE-ACS pt may show what ECG change
Nondiagnostic T-wave & nondiagnostic ST-segment changes
Extracorporeal cardiopulmonary resuscitation (ECPR) might help tx what c/o cardiac arrest
OD - PE - Hypothermia
Critical thinking uses
Obtaining an initial impression, determining course of action, anticipating roles & functions as part of team based on pts presentation/condition, consistently re-evaluating situation for changes, interpreting changes & applying them to pts care & tx, & modifying actions based on changes you observe.
When obtaining 12-lead ECG, where should electrodes for limb leads be placed?
On upper arms and on the thighs/calves Avoid bony areas like the ankles
STEMI mgmt focuses on early reperfusion therapy with
PCI or Fibrinolytic therapy
78yo pt who had knee replacement sx 2d ago goes into cardiac arrest. What underlying cause should be top concern
PE
ECG rhythm strip part that's time from beginning of atrial depolarization to beginning of ventricular depolarization
PR Interval
Pt in cardiac arrest experiences ROSC. As part of post-cardiac arrest care, pt is receiving mechanical ventilation. Finds that would indicate need for change in vent settings to optimize pt ventilation & oxygenation
PaCO2 35 - SaO2 92% - ETCO2 50
Don't place AED pads over___________
Pacemaker
ROSC Clinical indications
Palpable pulse, measurable BP, ETCO2 value > 40 mmHg - Regular rate/rhythm not necessarily indication
Sudden Interruption in Pulmonary Perfusion
Partial disconnect/cuff leak appearance - Cardiac arrest, massive EBL d/t surgeon cutting something big, massive PE, or surgeon compressing a major vessel
Factors to consider when determining whether to continue resuscitation effort
Physiologic data - Underlying c/o arrest - Health prior to arrest - Duration of resuscitation effort - Initial arrest rhythm (shockable rhythm = better outcome) - Witness vs unwitnessed arrest - Time to start CPR & time to defib - Pre-arrest state - Whether ROSC achieved at some point
Pt in cardiac arrest, resuscitation team initiates ventilations via BVM resuscitator. Development of which condition during provision of care would lead team to suspect that improper technique being used
Pneumothorax
Colorimetric Analysis Pros
Portability - Low cost - Applicable outside OR - No need for other equipment - Color change reversible & can change from breath to breath
Hyperventilation/Overbagging SE
Potential adverse hemodynamic effects - Increased Intrathoracic pressure - Decreased CO - Decreased Cerebral flow
Waveform indicates
Presence of CO2 - Even abnormal waveform is indication CO2 is present
Tx for pt w. imaging revealing hemorrhage depends on cause & severity of bleeding. In addition to BLS measures, care should include what else
Prevent seizures - Controlling internal bleeding - Decreasing ICP - Decreasing BP
Concept that helps to promote effective/efficient teamwork & reduces likelihood of errors is called _____.
Problem solving
Assess
Process of gathering data to help determine whats happening. To ensure most pressing problems addressed first, take phased, systematic approach to assessment: perform rapid assessment, primary assessment & when condition allows, secondary assessment. In emergency situation, assessment ongoing.
Survival advantage of providing CPR to pt in VFib
Produces small amount of blood flow to heart
Goals for Pt Mgmt During Post-Cardiac Arrest Period
Promoting neurological recovery, optimizing ventilation/oxygenation, managing hemodynamics, correcting cardiac instability, & addressing underlying c/o cardiac arrest
Resuscitation team decides to place advanced airway in pt whose in cardiac arrest. How should ventilations be provided following placement of airway?
Provide 1 ventilation q6s w/o pausing compressions
Which assessment could be made during rapid assessment?
Pt is diaphoretic
Respiratory baseline that slopes upward & increases w. each breath suggests
Pt rebreathing CO2
On rapid assessment, you note increased work of breathing, AEB tripod positioning, inability to speak > 1-2 words at a time & diaphoresis. What assessments should you obtain during primary assessment?
Pulse ox & VS
CO2 Monitor Detects Complete Airway Obstruction & Extubation Faster Than
Pulse ox & VS monitoring
Appropriate tx as part of primary assessment of pt w. suspected ACS
Pulse ox - Vascular access - EKG - Cardiac monitoring
What parts of heart's electrical conduction system play role in ventricular contraction? (3)
Purkinje fibers - Bundle of His - Bundle Branches
In 3rd-degree AV block, there is no electrical communication btw atria & ventricles, so no relationship btw P waves & the _________
QRS Complexes
What increases chance of successful conversion of VFib
Quality compressions immediately before defibrillation
What is rapid assessment?
Quick assessment to ensure safety, form initial impression of pt, & if pt appears to be unresponsive check for responsiveness/breathing/pulse
HCP is establishing cardiac monitoring w. 5 electrode system & demonstrates proper use by placing green electrode on
RLQ
Pt suddenly collapses sitting in healthcare facility. HCP sees & runs over to assess. 1st assessment performed
Rapid assessment
In 6-person high-performance resuscitation team, which tasks are responsibilities of team members? (3)
Recoding key data, sharing pertinent observations, & performing chest compressions
Which complications can occur when providing ventilations w. BVM resuscitator?
Regurgitation/Aspiration (d/t gastric insufflation) - Tension Pneumothorax (d/t barotrauma) - Decreased Cardiac Output (d/t decreased venous return)
Mr. Hernandez has a pulse and is making an effort to breathe but is still unresponsive. The monitor shows normal sinus rhythm with a rate of 80 bpm. Mr. Hernandez's vital signs are as follows:Blood pressure: 128/80 mmHgHeart rate: 80 bpm, radial pulses presentRespirations: 9 breaths/minSpO2: 90%ETCO2: 60 mmHgWhat should the team do next
Request laboratory studies to assist in evaluating perfusion status. Obtain a 12-lead ECG. Administer supplemental oxygen at a rate of 10-15 liters/min. Support ventilations at a rate of 10 to 12 breaths/min to lower the end-tidal carbon dioxide (ETCO2) level to 35-40 mmHg.
Respiratory compromise manifests along continuum. When pt is breathing but respiratory system unable to meet metabolic demands, pt is in:
Respiratory Failure
Pt in ED is in respiratory compromise. Team is monitoring pt w. capnography, ETCO2 initially 33 & later 40. From readings, team identifies pt is progressing in what stage of respiratory compromise
Respiratory distress - Capnography can objectively assess severity of respiratory distress - Early on, pt will often hyperventilate = Hypocapnia reflected by low ETCO2 (< 35) - As distress increases, & pt begins to tire ETCO2 may return to normal (35-45) - If respiratory failure, ETCO2 increases > 45 = Hypoventilation
Assessment of pt reveals respiratory compromise. From assessment, team identifies pt in earliest stage of condition. Earliest stage
Respiratory distress - Occurs along continuum - Respiratory distress Respiratory failure Respiratory arrest
Assessment shows ETCO2 55 & SaO2 88%. HCP would interpret findings as indicative of
Respiratory failure
Compressor
Responsible for chest compressions
Data Manager
Responsible for communicating & recording key data during resuscitation effort (i.e. data r/t med admin & interruptions to chest compressions)
Medication Administrator
Responsible for establishing vascular access & administering medications.
AED/Defibrillator Operator
Responsible for managing AED or defibrillator & establishing any other monitoring. Also, relieves team member performing compressions
Airway manager & ventilator
Responsible for managing airway & providing ventilations. Trained RT, if available, would fill role
Team Leader
Responsible for prioritizing & directing other team members' actions.
Colorimetric Analysis Con
Results semiquantitative, provide results for range instead of a definite number
Mr. Hernandez remains in pulseless ventricular tachycardia. What is the appropriate next intervention
Resume CPR, administer one shock and administer amiodarone.
After one shock and 2 minutes of CPR, the rhythm check reveals no change in the rhythm. What is the appropriate next intervention
Resume CPR, administer one shock and administer epinephrine.
Rapid Assessment S/S of Respiratory Compromise
Retractions - Use of accessory mm to breathe - Tripod positioning - Inability to speak in complete sentences - Pale/Ashen/Cyanotic skin - Diaphoresis - Restlessness/Agitation - Altered LOC
MI S/S
Retrosternal CP - Dyspnea - N/V - Fatigue/Weakness - Loss of Consciousness
Myocardial Stunning
Reversible reduction of function of heart contraction after reperfusion not accounted for by tissue damage or reduced blood flow - Even though heart is perfusing again, it's not working very well as pump
Part of secondary assessment of pt in cardiac arrest
Reviewing medical hx
Contraindication to Nitro for mgmt of ACS
Right ventricular infarction & dysfunction
Besides MI, what are life-threatening c/o CP
Ruptured esophagus - Perforating PUD - Pneumothorax - Aortic dissection - PE
ECG changes that suggest ischemia
ST-segment depression & T-wave inversion
ECG findings expected in pt w. high-risk NSTE-ACS
ST-segment depression & T-wave inversion in 2+ contiguous leads
ECG findings suggesting NSTE-ACS
ST-segment depression, T-wave inversion & transient T-wave elevation
12-lead ECG reveals tachyarrhythmia w. narrow QRS complexes (< 0.12s). You would classify this arrhythmia as ________.
SVT
15-lead ECG Indication
Screen for posterior MI if ACS suspected but 12-lead doesn't show ST elevation or for right ventricular infarction if 12-lead reveals evidence of inferior wall MI (II, III, aVF) - Suspicion of posterior wall/right ventricular involvement
Diagnostic studies useful for identifying c/o cardiac arrest
Serum electrolytes - Bedside US - CXR
Responsibilities of team leader? (3)
Sets clear expectations, leads debriefing, assigns roles
Bronchospasm Capnography
Shark fin wave - Asthma, blocked tube, COPD/obstructive disease, upper airway obstruction
EKG: Heart's electrical conduction system part that generates electrical impulses initiating heartbeat rhythm/rate:
Sinoatrial (SA) node
This is the rhythm that the telemetry technician observed on the cardiac monitor at the central telemetry station. What is your interpretation of the rhythm on the cardiac monitor
Sinus Bradycardia
Torsades de pointes may revert to which rhythm
Sinus Rhythm - VFib - Pulseless VTach
Arrhythmias that are narrow-complex tachyarrhythmias? (3)
Sinus tachycardia - AFlutter - AFib
What should the team do now
Stop CPR and check for a pulse.
Catastrophic Loss of Ventilation
Sudden CO2 drop - Extubation, circuit disconnect, vent failed, blocked ETT, plugged/kinked sample line, or migration to esophagus
Pt has ROSC after cardiac arrest & healthcare team is conducting secondary assessment to determine possible c/o pt arrest. Hx reveals that before arrest, pt exhibited JVD, cyanosis, apnea, & hyperresonance on percussion. Pt difficult to ventilate during response. Team most likely suspected
Tension pneumothorax
Differential dx conditions for pt w. s/s of acute stroke
Systemic infx - Seizure disorder - Hypoglycemia - Migraine
What part of ECG rhythm strip represents repolarization of ventricular myocardial cells?
T-waves
To promote neurologic recovery in comatose post-cardiac arrest pt, which tx is appropriate
TTM (targeted temperature mgmt), should be considered for any pt unable to follow verbal commands following ROSC
Mr. Hernandez remains unresponsive to verbal commands. What therapy should the team initiate to promote his neurological recovery
Targeted temperature management (TTM)
The team decides to intubate Mr. Hernandez. How does this affect compressions and ventilations
The team should provide ventilations at a rate of 1 ventilation every 6 seconds without pausing compressions.
You need to set up cardiac monitoring w. 5-electrode system. Where should you place white electrode?
Top right chest anatomically oriented
Chest Compression Fraction
Total percentage of time during resuscitation attempt in which active chest compressions are being performed. Needs to be >= 60% w. goal of 80
(T/F) Underlying c/o primary PEA can be r/t inadequate volume, impaired myocardial contractility or both
True
When assessing pt w. respiratory compromise, it's important to determine where pt is on continuum of respiratory compromise. T/F
True
Key decision point when providing care for pt w. stroke is determine
Type of stroke
Prolonged respiratory upstroke that isn't vertical indicates
Uneven alveolar emptying d/t bronchospasm
Rhythm that requires synchronized cardioversion
Unstable SVT
Pt has mild to moderate recurrent CP wo N/V. ECG shows ST depression w. transient T-wave elevation indicative of NSTE-ACS. Cardiac markers obtained & aren't elevated. Pt risk-stratification score indicates low risk. Findings suggest
Unstable angina
Three minutes after the initial dose of atropine is administered, Mr. Hernandez's heart rate has increased slightly to 34 bpm, but there is no change in his clinical condition. How should Dr. Hudson direct the team's next actions
Valerie, please administer a second dose of atropine. Haley, please attach defibrillator/pacing pads.
Defining characteristics of stroke
Vascular cause - Lasting neuro deficit - Sudden onset - Primary involvement of CNS
Conditions that contribute to pathophysiology of ACS
Vasospasm - Development of occlusive intracoronary thrombus - Transient platelet aggregation - Plaque erosion
What has the rhythm changed to
Ventricular tachycardia
EKG of pt c/o dizziness, syncope, SOB = Sinus brady. When reviewing med hx, HCP identifies what med(s) as potential cause of condition
Verapamil - Digoxin - Metoprolol - Meds associated w. sinus brady include BB (metoprolol) & CCB (verapamil/digoxin)
Pt presents to ED w. suspected ACS. EKG & cardiac biomarkers indicate a STEMI. Exam reveals s/s of left ventricular dysfunction. S/S to support this
Weak peripheral pulses - Hypotension - Crackles
Hyperkalemia suspected c/o cardiac arrest, confirmed on ECG d/t
Wide-complex ventricular rhythm & tall, peaked T waves
In 12-lead ECG, the 4 limb leads produce views in frontal plane. What are those views? (6)
aVL - I - aVR - II - III - aVF