Quiz 9

A single bout of resistance exercise training increases muscle protein synthesis by as much as ______% above resting levels?

120-150

High responders to endurance exercise training (i.e., individuals that achieve large increases in VO2 max) can achieve up to______% improvement in VO2 max following a prolonged and intense training program.

50

In theory, concurrent resistance and endurance exercise training can impair muscle protein synthesis following a bout of resistance exercise. The proposed mechanism to explain this inhibition is_____________________________________.

AMP kinase activation of TSC2

T/F Following endurance training, maximal cardiac output during exercise is increased due to an increase in the thickness of the left ventricular wall (i.e, ventricular muscle mass increases).

F

Several mechanisms have been proposed to explain why concurrent endurance and resistance (strength) training impedes strength development compared to strength training alone. Which of the following mechanisms have NOT been proposed to explain why concurrent training impedes strength development? neural factors overtraining depressed muscle protein synthesis None

None

T/F Following endurance training, afterload decreases during maximal exercise because there is a decrease in sympathetic vasoconstrictor activity in the arterioles of the exercising muscles.

T

T/F In a "two-legged" maximal cycle ergometer test, if each leg were to vasodilate to the extent experienced in a one-legged VO2 max test, mean arterial blood pressure would fall below normal levels.

T

Following an acute bout of endurance exercise training, the increase in mitochondrial biogenesis occurs within _____ following the completion of the exercise session.

hours

Which of the following endurance training adaptations does NOT result in lower lactate production in the contracting muscles? increased glycogen utilization increased H4 form of LDH increased mitochondrial uptake of pyruvate decreased pyruvate formation

increased glycogen utilization

Which of the following physiological factors does NOT contribute to the endurance exercise training-induced increase in the maximal a-vO2 difference? increased mitochondria volume in the muscle increased capillaries in the muscle increased hemoglobin in the blood increased muscle blood flow

increased hemoglobin in the blood

Which of the following factors does NOT contribute to the endurance exercise training-induced improvement in stroke volume during exercise? increased end-diastolic volume increased peripheral resistance increased cardiac contractility decrease in total peripheral resistance

increased peripheral resistance

After a 15-week program of endurance exercise training, which of the following cardiovascular variables does NOT decrease after 14 days of detraining? maximal heart rate VO2 max maximal cardiac output maximal stroke volume

maximal heart rate

It is well known that resistance exercise training promotes an increase in muscle protein synthesis by activating the mechanistic target of rapamyosin (mTOR) which results in increased protein synthesis. Recent evidence suggests that two signaling molecules interact to directly activate mTOR. These molecules are:

phosphatidic acid and Ras homologue enriched brain (Rheb)

Cross-sectional studies demonstrate that the physiological variable responsible for the large variation in VO2 max across the normal population is maximal

stroke volume

In resting skeletal muscle fibers, Ras homologue enriched brain (Rheb) activation of mTOR is inhibited by ______.

tuberous sclerosis complex 2 (TSC2)