Bone Fractures and Bone Remodeling
Wolf's Law
a bone grows or remodels in response to the forces or demands placed upon it
Hematoma formation
a mass of clotted blood forms at fractured site, site becomes swollen, painful, and inflamed
Displaced fracture
bone ends are out of normal alignment
Simple fracture (closed)
bone ends do not penetrate the skin
Compound fracture (open)
bone ends penetrate the skin
Non-Displaced fracture
bone ends retain normal position
Comminuted fracture
bone fragments into three or more pieces; common in the elderly
Complete fracture
bone is broken all the way through
Compression fracture
bone is crushed; common in porous bone (vertebrae, wrist)
Incomplete fracture
bone is not broken all the way through
Depressed fracture
broken bone portion pressed inward; typical skull fracture
Epiphyseal fracture
epiphysis separates from diaphysis along epiphyseal line (along growth plate); occurs when cartilage cells are dying
Bone homeostasis
every week we recycle 5-7% of our bone mass, spongy bone is replaced every 3-4 years, compact bone is replaced approximately every 10 years
Bone remodeling
excess material on bone shaft exterior and its the medullary canal is removed, compact bone is laid down the reconstruct shaft walls
Bony callus formation
fibrocartilaginous callus converts into a bony (hard) callus, bone callus begins 3-4 weeks after injury, and continues to firm union is formed 2-3 months later
Fibrocartilage callus
forms when osteoblasts and fibroblasts migrate to the fracture and begin reconstructing the bone
Stages of healing a bone fracture
hematoma formation, fibrocartilage callus, bony callus formation
Greenstick fracture
incomplete fracture where one side of the bone breaks and the other side bends; common in children
Bone fractures are classified by:
position of bone ends after fracture, the completeness of the break, the orientation of the bone to the long axis, whether or not the bones ends penetrate the skin
Spiral fracture
ragged break when bone is excessively twisted; common sports injury
Hormonal mechanism
rising blood Ca2+ levels trigger the thyroid to release calcitonon, falling Ca2+ levels signal the parathyriod glands to release PTH
PTH
signals osteoclasts to degrade bone matrix and release Ca2+ into the blood
Calcitonin
simulates calcium salt deposit in bones
Linear fracture
the fracture is parallel to the long axis of the bone
Transverse fracture
the fracture is perpendicular to the long axis of the bone