CHAPTER 13 - EX PHYS

A single bout of resistance exercise training increase muscle protein syntheses by as much ___% above resting levels? 10-20 50-60 120-150 200-300

120-150

Following a single bout of resistance exercise training, muscle protein synthesis can remain elevated above baseline for as much as__ in both trained individuals 10-15 hours 15-20 hours 20-25 hours 40-50 hours

40-50 hours

Heritability (i.e., genetics) determines approximately__% of VO2 max variability in sedentary adults? 25 35 50 90

50

In theory, concurrent resistance and endurance exercise training can impair muscle protein synthesis following a bout resistance exercise. The proposed mechanism to explain this inhibition is? AMP kinase activation of TSC2 AMP kinase activation of mTOR AMP kinase activation of PGC-1a P38 activation of TSC2

AMP kinase activation of TSC2

The enhanced capacity of the trained muscle to use fatty acids as a fuel results in? Increase uptake of fatty acids Decrease utilization of muscle glycogen Sparing of blood glucose All if these answers are correct

All if these answers are correct

Following endurance training, maximal cardiac output during exercise is increased due to an increase in the thickness of the left ventricular wall (i.e., ventricular muscle mass increases). True false

false

The initial decrease in VO2 max with detrain occurs because skeletal muscle adaptation (a-VO2 difference) are lost True False

false

Endurance training has been shown to reduce the oxygen deficit in subjects performing a submaximal exercise. This is due to? An increase maximal cardiac output Increase in the number of mitochondria and capillaries An increase heart rate A greater anaerobic capacity

Increase in the number of mitochondria and capillaries

Which of the following endurance training adaptation assist in maintaining acid- base balance during exercise? Increase mitochondrial volume in skeletal muscle Increase M4 form of LDH Increase lactate production Increase pyruvate formation

Increase mitochondrial volume in skeletal muscle

Which of the following endurance training adaptation does NOT result in lower production in the contracting muscles? Increased glycogen utilization Increased H4 form of LDH Increased mitochondrial uptake of pyruvate Decreased pyruvate formation

Increased glycogen utilization

Which of the following factors contributed to the endurance exercise training-induced improvement in VO2 max? Increased end diastolic volume Increased maximal stoke volume Increased maximal heart rate None of these answers is correct

Increased maximal stoke volume

Endurance training has been shown to reduce the oxygen defict in subjects performing submaximal exercise. This is due to? An increased maximal cardiac output Increases in the number of mitochondria and capillaries An increased heartrate. A greater anaerobic capacity

Increases in the number of mitochondria and capillaries

After a 15- week program of endurance exercise training, which of the following cardiovascular variable does NOT decrease after 14 of detraining? Maximal heart rate VO2 max Maximal cardiac output Maximal stroke volume

Maximal heart rate

During the first 10 weeks of a resistance training program, the initial increase in muscular strength are due primarily to? Neural adaptations Muscle fiber hypertrophy Muscle fiber hyperplasia Both muscle fiber hypertrophy and muscle fiber hyperplasia are correct

Neural adaptations

It is well known that resistance exercise training promotes an increase in muscle protein syntheses by activating the mechanistic target of rapamyosin (mTOR) which result in increased protein synthesis. Recent evidence suggests that two signaling molecules interact to directly activate mTOR_These molecule are? IGF-1 and Akt IGF-1 and phosphatidic acid IGF-1 and Ras homologue enriched brain (Rheb) Phosphatidic acid and Ras homologue enriched brain (Rheb)

Phosphatidic Acid and Rheb

Endurance training results in increase mitochondrial and capillary density in muscle but has no effect on muscle glycolytic capacity. This is an example of what training principle? Overload Specificity Reversibility None of these

Specificity

Endurance training results in increased mitochondrial capillary density in muscle but has no effect on muscle glycolytic capacity. This is an example of what training principle? Overload Specificity Reversibility None of these

Specificity

Cross-sectional studies demonstrate that the physiological variable responsible for the large variation in VO2 max across the normal population is maximal? Heart rate Stoke volume Arteriovenous O2 difference Systolic blood pressure

Stoke volume

The increase in VO2 max that occurs following 4 months of endurance exercise training results from an increase in both maximal cardiac output and maximal a-VO2 difference. Relatively short duration of endurance exercise training (e.g., 1-4 month) increase VO2 max primarily by increasing? The maximal a-Vo2 difference The maximal cardiac output The maximal heart rate Both the maximal cardiac output and the maximal heart rate are correct

The maximal cardiac output

In resting skeletal muscle fibers, Ras homologue enriched brain (Rheb) activation of mTOR- is inhibited by? Phosphatidic acid Tuberous sclerosis complex 2(TSC2) IGF-1 None of these answers is correct

Tuberous sclerosis complex 2 (TSC2)