final review
how dos cardiac output at a given sub-max work rate adapt
does not really change, just below
which component of cardiac output is responsible for increased max Q
increased SV??
which of the following is NOT a factor of enhanced blood flow to muscle after training
increased blood pressure
what off-sets the decrease in resistance due to increased capillaries to maintain flow
increased blood volume
the respiratory pump increases blood flow (and stroke volume) by
increasing venous return (preload)
which of the following statements about muscular endurance is FALSE
it is closely related to cardiorespiratory endurance
which the following is FALSE regarding cardiac hypertrophy
it is typically accompanied by decreased ventricular volume
the presence/abundance of __ drives the negative feedback loop -inhibiting glycolysis
lactic acid
at which intensity is heart rate not impacted by aerobic endurance training
maximal
which of the following is FALSE regarding aerobic endurance training adaptations
metabolic economy increases (decreased O2 at same speed)
why doesn't blood flow through the coronary arteries during ventricular systole
the flaps of the aortic semilunar valve blocks the arteries
one of the primary drivers are cardiovascular drift
thermoregulation
an increase in parasympathetic (vagal) tone will result in
decreased resting heart rate
which of the following is TRUE regarding SUBMAXimal endurance capacity
submax is more closely related to competitive performance
why does it take longer to return to resting levels following exercise
2 of these: waste (CO2) removal continues after exercise ends/??
stroke volume for this subject increased by 40%, to what extent did max performance (work rate) improve
20%: from 18 to 22 km/h
which of the following forms of interval training likely were incorporated into the training regimen
5 x 800m; 15 sec rest
what is the limiting factor for aerobic capacity
CV system delivery and availability
during exercise, how does blood pressure change with increased intensity
SBP increases and DBP doesn't change
why is there a decrease in heart rate (from pre to post) at the same work rate
SV increases
why does the decreased diffusion gradient cause reduced flow into tissues? Flow = change in pressure/ resistance
absolute pressure gradient decreases
which of the 4 factors of stroke volume does NOT increase following endurance training
afterload (aortic pressure)
which component(s) of stroke volume experience positive adaptation through aerobic training
all of these: preload/ ventricular volume and strength/ afterload
because the body doesn't store oxygen, we can estimate aerobic ATP production by measuring
all of these: respiratory exchange expired oxygen volumes expired carbon dioxide volumes
blood flow =
blood pressure gradient/vascular resistance
why is RER lower for fat metabolism (oxidation)
fat requires more oxygen to fully metabolize
which of the following blood pressures is NOT affected following endurance training
healthy subject resting BP
max heart rate is
highest heart rate achieved during all-out effort
how does the CV system, during cardiovascular drift, change to aid thermoregulation
increased heart rate
though enzyme activity increases, most anaerobic improvements can be attributed to
increased muscular strength
capillary-to-fiber ratio is an effective measure of which blood flow adaptation
increased number of muscle capillaries
only by looking at the graph, which of the following training adaptations have occurred
increased stroke volume increased aerobic power
which of the following is MOST responsible for decreased ESV
increased ventricular muscle mass
sport specific adaptations support the theory that adaptations are
muscle specific
is pulmonary ventilation the MOST LIKELY limitation to maximum performance
no
what affect does oxygen have on ATP-PCr system
oxygen has no impact on ATP-PCr reactions
what causes gases to flow across the respiratory membrane
partial pressure gradients
why does blood flow
pressure gradients
the final product of glycolysis (for glycogenolysis) is
pyruvic acid
how do left ventricle changes differ in resistance and endurance athletes
resistance athletes don't increases LV volume
what is the main reason for the arterial-venous pressure differential
resistance of the vessels