Patho 2 Exam 3
Which client is at risk for developing a type II, antibody-mediated hypersensitivity reaction caused by complement-activated cellular destruction?
An Rh-negative mother who produces anti-D antibodies when the fetus is Rh-positive
A client is in the emergency department with a severe, type I IgE-mediated hypersensitivity reaction. What pharmacologic treatment should the nurse prepare to administer in this acute phase? Select all that apply.
An antihistamine Systemic corticosteroids Epinephrine
A nurse is caring for a client who has systemic lupus erythematosus (SLE). Which of the following causes the disease?
Autoimmune process
Contact with poison ivy has resulted in intense pruritus, erythema, and weeping on a client's forearm. Which pathological process is responsible for the client's manifestations?
Cytokine release by sensitized T cells
The nurse is assessing a client in early stage of rheumatoid arthritis. Which assessment findings would the nurse expect to find? Select all that apply.
Erythema Swelling Warmth Tenderness
Type III hypersensitivity immune responses can be harmful when immune complex deposits in tissue cause which type of damage to tissues?
Inflammation
Which assessment findings will help the nurse to confirm the diagnosis of rheumatoid arthritis? Select all that apply.
Joint stiffness Finger and hand pain Wrist pain Limited joint mobility
The type of hypersensitivity reaction that is dependent on IgE-mediated activation of mast cells and basophils and the subsequent release of chemical mediators of the inflammatory response is known as which of the following types of hypersensitivity reaction?
Type I
The nurse is teaching a client with rheumatoid arthritis about pannus, which develops in the affected joint area. What does the nurse include to describe pannus?
Vascular granulation tissue that destroys cartilage and bone
Which diagnostic finding has been strongly linked to systematic lupus erythematosus (SLE)?
elevated anti-nuclear antibodies (ANA)
